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Posters ration of products with high yields without the formation of secondary products that sometimes are very difficult to remove. In this study is described the influence of the biocatalyst on the transesterification of grape seed oil with solketal and glycidol. Two own catalysts, prepared from resting-cells of Aspergillus flavus and Rhizopus oryzae, and five commercial enzymes, Amano Lipase AYS Candida rugosa ; Aldrich ; , Novozym 435 Novo Nordisk ; , Amano Lipase A Aspergillus niger ; Aldrich ; , Amano Lipase PS Pseudomonas cepacia ; Aldrich ; , Lipozyme, immobilized Mucor miehei ; Fluka ; , were used to carry out this reaction. In a 12 vial were added 300 mg of grape seed oil, 135 mg of solketal, 30 mg of biocatalyst and 4 ml of isooctane. The mixture was then magnetically stirred at 50 C for 196 h. Samples were taken at 24, 48, 72, and 196 h and analyzed by HPLC-RP. Similar experiments were carried out using glycidol instead of solketal as reagent for the transesterifiaction of the vegetable oil. Among the enzymes assayed, Rhizopus oryzae gave the best results. Thus, the solketal derivative yield was 11 mg.ml1 at 72 h reaction. Although the same biocatalyst gave the highest transformation when glycidol was used, the yield was much lower. REFERENCES 1. Garca-Alles L. F., Gotor V.: Biotechnol. Bioeng. 59, 684 1998 ; . 2. Basheer S., Mogi K., Nakajima M.: Am. Oil Chem. Soc. 72, 511 1995 ; . 3. Gandolfi R., Gualandris R., Zanchi C., Molinari F.: Tetrahedron: Asymmetry 12, 501 2001. Ment of chronic neck pain. While advancing a 25-gauge Quincke needle at the C interspace, leakage of spinal fluid was noted. The needle was withdrawn until no leakage was evident and then 5 mm further. Then, methylprednisolone 80 mg diluted to 8 ml normal saline was given extradurally. Two days later she developed headache that was located posteriorly and exacerbated by the upright position. She was told to avoid the upright position, to drink plenty of fluids, and to take acetaminophen 300 mg po every four to six hours. Her headache did not respond and four days after the dural puncture she was instructed to take Cafergot tablets ergotamine tartrate 1 mg and caffeine 100 mg ; 200 mgpo tid. This regimen was continued for 24 hr but did not provide relief. The patient was referred for further evaluation and admitted to the hospital. Her physical examination was unremarkable. A midline scar was present in the cervical area. There were no visual or auditory disturbances. Her headache was still posteriorly located and constant. It improved dramatically in the recumbent position and worsened in the sitting position. She reported that she had never experienced this kind of headache and described it as unbearable. Four months previously she had undergone cervical discectomies with no relief of her neck pain, which was still confined to the posterior neck area and rated mild at that time. Her oldest MRI showed herniated discs at C5 6 and Q.7 and a more recent one showed signs of cervical discectomies. Under sterile conditions and in the sitting position a lumbar extradural blood patch was performed at the L3 4 interspace with 20 ml autologous blood via a 17-gauge epidural Tuohy needle. Her headache resolved within one to two minutes of the completion of the procedure and she felt comfortable in maintaining the sitting position. One week after the procedure, and 12 days after her dural puncture, the patient was free of headache. Discussion Extradural blood patching is used to treat PDPH consequent to a lumbar puncture.2 The continuous leakage of CSF from the dural hole and the reduction in CSF pressure seems to be the responsible mechanism of PDPH. Indeed the incidence of PDPH is directly related.

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1989 Mar; 6 1 ; : 1-6. Clinical benefits of early cold therapy in accident and emergency following ankle sprain. Sloan JP, Hain R, Pownall R. Accident and Emergency Department, University Hospital, Nottingham, England. One hundred and forty-three patients presenting with ankle sprains within 24 h of injury were entered into a double blind study. Treatment consisted of a standardized regime of high dose non-steroidal anti-inflammatory medication andan elastic support for all patients, who were then randomly allocated to twogroups. One group received immediate cold therapy, the other received simulated therapy. Assessments made at 7 days showed a trend in favour of the groupreceiving cold therapy, although this did not reach significance. It isconcluded that cold therapy continued.
Peritransplant F Ab ; 2-IT days 0 and 1 plus DSG days 0 14. Methylprednisllone was given to all groups in a tapered dose over days 0 2, with no other immunosuppression. Rejection; surgical gastrointestinal ; complication after cannulation of hepatic vessels and intrahepatic glucose tolerance test died euglycemic. Guidelines A. For the treatment of the first acute cellular rejection episode, high doses of intravenous methylprednisolone are recommended. This treatment is expected to reverse most acute rejection episodes. Although the use of polyclonal ATG ALG ; or monoclonal OKT3 ; antibodies as first-line therapy is effective, their adverse event profile and cost mean that the use of corticosteroids as first-line therapy is preferred. Evidence level C ; B. ATG ALG or OKT3 are recommended for the treatment of severe acute rejection episodes Banff grade III ; , recurrent acute rejection episodes, corticosteroidresistant rejection episodes or in patients with contraindications to corticosteroids. Evidence level C ; C. In patients with recurrent rejection after anti-T lymphocyte antibody treatment, it is recommended to modify baseline immunosuppression. Evidence level B.

The present guidelines draw on the results of the Round Table, a summary of which, prepared by a group of participating scientists, is available on the following Internet site: : europa .int comm research biosociety index. Table 12.3: Antibiotic resistance markers Consultations 1997 [1136] Crop antibiotic resistance Antibiotic resistance and metoprolol. Tell your doctor your medical history, especially if you have: heart disease, liver disease, kidney disease, blood pressure problems, any allergies. L. Dunkley and A. S. M. Jawad function tests showed a restrictive defect with reduced transfer factor; a high resolution CT scan of the chest demonstrated bilateral basal ground glass shadowing. The prednisolone dose was increased to 20 mg twice daily, 3weeks after admission. The facial and upper limb oedema responded over a 1-week period. The proximal muscle weakness however rapidly progressed such that the patient had difficulty sitting up. Despite intravenous IV ; methylprednisolone 500 mg on three successive days, the patient's clinical condition continued to deteriorate and she was given one pulse of IV cyclophosphamide 500 mg on day 51. Nine days later, the patient developed a fever and respiratory failure requiring intubation and transfer to the intensive care unit. Repeat chest X-ray revealed bilateral interstitial infiltrates to the mid-zones; bronchoscopy with bronchoalveolar lavage confirmed a diagnosis of Pneumocystis carinii pneumonia. There was no evidence of respiratory muscle weakness. Despite aggressive antimicrobial therapy and continuation of prednisolone, the patient had a prolonged ITU admission with recurrent infections and unsuccessful attempts to wean her from respiratory support. She eventually passed away 4 months after her initial admission and miacalcin.
Manju said that since she was very young at the time of hysterectomy, everyone, especially her husband, had advised her to opt for HRT. In retrospect, in spite of some recent studies which reported increased morbidity associated with long term use of HRT, Manju feels that opting for HRT treatment was the right thing to do in her case. Interpretation of findings and discussion Although the four women who were interviewed for the case study were different in many respects, certain factors emerge common to all the cases. All of them suffered from various physical and psychological problems both before and after the hysterectomy. In the first three cases, severe, prolonged and irregular bleeding had interfered with the daily functioning of those women forcing them to opt for hysterectomy. The common complaints during the post-hysterectomy period were hot flushes, irritability, mood swing and weight gain. Other problems varied from person to person. In terms of sexual life, responses varied from loss of libido to increase in sexual pleasure after the operation. It was seen that even in those cases where heavy and painful bleeding led to the hysterectomy, the women undergoing the surgery had mixed feelings about it. While they accepted the hysterectomy as necessary and expressed relief from painful symptoms after the hysterectomy, they had approached surgery with various apprehensions. Such apprehensions had its origin in a ; inadequate information given to the women by the doctor, and b ; accounts of other people regarding what to expect after the hysterectomy. It was seen that the accounts of post-hysterectomy life passed on to women who are to undergo hysterectomy by friends, relatives and acquaintances are mostly of a negative character. Such accounts make the patients apprehensive and anxious. In addition, the information given by the health care providers to the women undergoing hysterectomy was found to be inadequate. In most cases, doctors did not discuss the posthysterectomy problems or its treatment with these women.
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Three trials have compared the relative efficacy of IV methylprednisolone and ACTH treatment in MS relapses Abbruzzese et al., 1983; Barnes et al., 1985; Thompson et al., 1989 ; . One study including 14 patients treated with IV methylprednisolone 1 g daily for 7 days ; and 11 patients treated with intramuscular IM ; ACTH 80 units, 60 units, 40 units, and 20 units daily, each for 1 week ; reported more rapid improvement after 3 and 28 days ; after IV methylprednisolone treatment than after ACTH treatment, but there was no significant difference after 3 months Barnes et al., 1985 ; . The patient blinding and the primary outcome were not clearly defined, however, for which reason this study should be considered a class III study. One class II study compared the administration of 1 g methylprednisolone once daily for 3 days to ACTH treatment 80 units for 7 days, 40 units for 4 days, and 20 units for 3 days ; in 61 patients, and found no difference in terms of rate of recovery or final outcome after 12 weeks Thompson et al., 1989 ; . A class III study including 60 patients, treated with either IV methylprednisolone 20 mg kg, day 13; 10 mg kg, day 47; 5 mg kg day, 810; and 1 mg kg, day 1115 ; or ACTH 1 mg IV daily for 15 days ; , also did not provide evidence of any difference in the efficacy of ACTH and methylprednisolone treatment Abbruzzese et al., 1983 ; . These three studies found no major differences in adverse events between methylprednisolone and ACTH treatment. Thus there is no evidence of any major difference in the efficacy of ACTH and methylprednisolone treatment from comparative studies, but the clinical trials were too small to rule out some difference in efficacy. Indeed, in the Cochrane review it was suggested that methylprednisolone treatment could still confer greater benefit than treatment with ACTH, and the administration of methylprednisolone is simpler than the more prolonged treatment with ACTH Filippini et al., 2000 ; . In a separate meta-analysis Miller et al., 2000 ; of three double-blind, randomized, controlled trials comparing methylprednisolone treatment 500 mg or more daily ; to placebo, it was concluded that treatment with IV methylprednisolone 15 mg kg, day 13; 10 mg kg, day 46; 5 mg kg, day 79; 2.5 mg kg, day 1012; 1 mg kg, day 1315 followed by oral prednisone tapered slowly over 120 days; Durelli et al., 1986 ; , IV methylprednisolone without a tapering dose 500 mg once daily for 5 days; Milligan et al., 1987 ; , or oral methylprednisolone 500 mg once daily for 5 days followed by 400, 300, 200, and 8 mg once daily the subsequent 10 days; Sellebjerg et al., 1998 ; resulted in significantly faster recovery than did treatment with placebo Table 1 ; . The two first trials provided follow-up data in a placebo-controlled design and monopril. In close cooperation, the two companies will enhance their activities in providing promotional and scientific information to the medical community and will effectively promote the innovative feature of this product to medical professionals.
Thereby obtaining the beneficial effects of both types of preparations. Mixing the corticosteroid preparation with a local anesthetic is a common practice for avoiding the injection of a highly concentrated suspension into a single area. The anesthetic provides early relief of symptoms and helps confirm the diagnosis. Low-solubility agents, favored for joint injection, should not be used for soft tissue injection because of the increased risk of surrounding tissue atrophy. Methylprddnisolone Depo-Medrol ; is often the agent selected for soft tissue injection and morphine.
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And recommendation. The ODS Director and the Office of Pharmacoepidemiology and Statistical Science OPaSS ; Director also reviewed the consult. Both disagreed with the findings and recommendation. Solubilization, Purification, and Properties. J Biol Chem 1964; 239: 2379-2385. Lipscomb JD. Electron paramagnetic resonance detectable states of cytochrome P and naproxen. Life tables show numbers of patients and number of events every 6 months, for example, methylprednisoloe used for.
Generic Drug Name METHADONE 5 MG TABLET METHAZOLAMIDE 50 MG TABLET METHIMAZOLE 10 MG TABLET METHIMAZOLE 5 MG TABLET METHOCARBAMOL 500 MG TABLET METHOCARBAMOL 750 MG TABLET METHOTREXATE 2.5 MG TABLET METHOTREXATE LPF 25 MG ML VIAL METHYLDOPA 250 MG TABLET METHYLDOPA 500 MG TABLET METHYLPHENIDATE 10 MG TABLET METHYLPHENIDATE 20 MG TAB SA METHYLPHENIDATE 20 MG TABLET METHYLPHENIDATE 5 MG TABLET METHYLPRED 4 MG TAB DOSEPAK METHYLPREDNISOLONE 4 MG TAB METOCLOPRAMIDE 10 MG TABLET METOCLOPRAMIDE 5 MG TABLET METOCLOPRAMIDE 5 MG 5 SYRP METOLAZONE 2.5 MG TABLET METOLAZONE 5 MG TABLET METOPROLOL 100 MG TABLET METOPROLOL 25 MG TABLET METOPROLOL 50 MG TABLET METRONIDAZOLE 0.75% CREAM METRONIDAZOLE 250 MG TABLET METRONIDAZOLE 500 MG TABLET MEXILETINE 150 MG CAPSULE MIDAZOLAM HCL 1 MG ML VIAL MIDODRINE HCL 10 MG TABLET MIDODRINE HCL 5 MG TABLET MINOCYCLINE 100 MG CAPSULE and nasonex.
Pregnant patients taking this drug will only do danger for the fetus, because methlyprednisolone tablets side effects.
Methylprednisolone should be used cautiously if you have an underactive thyroid, liver cirrhosis, or herpes simplex virus ; infection of the eye and neurontin.
Rothman, S. M., and J. W. Olney 1986 ; Glutamate and the pathophysiology of hypoxic-ischemic brain damage. Ann. Neurol. 19: 105111. Rothman, S. M., and J. W. Olney 1987 ; Excitotoxicity and the NMDA receptor. Trends Neurosci. 10: 299-302. Sanchez-Prieto, J., and P. Gonzalez 1988 ; Occurrence ofa large Cal + independent release of glutamate during anoxia in isolated nerve terminals synaptosomes ; . J. Neurochem. 50: 1322-1324. Sasaki, T., T. Nakagomi, T. Kirino, A. Tamura, M. Noguchi, I. Saito, and K. Takakura 1988 ; Indomethacin ameliorates ischemic neuronal damage in the gerbil hippocampal CA, sector. Stroke 19: 13991403. Siesjo, B. K. 198 1 ; Cell damage in the brain: A speculative synthesis. J. Cereb. Blood Flow Metab. 1: 155-185. Siesjo, B. K., and F. Bengtsson 1989 ; Calcium fluxes, calcium antagonists, and calcium-related pathology in brain ischemia, hypoglycemia, and spreading depression: A unifying hypothesis. J. Cereb. Blood Flow Metab. 9: 127-140. Simon, R. P., J. H. Swan, T. Griffiths, and B. S. Meldrum 1984 ; Blockade ofN-methyl-D-aspartate receptors may protect against ischemit damage in the-brain. Science 226: 850-852: Smith. D. S. S. Rehncrona. and B. K. Siesio 1980 ; Barbiturates as protective agents in brain'ischemia and as free radical scavengers in vitro. Acta Physiol. Stand. Suppl. 492: 129-134. Smith. M.-L. R. N. Auer. and B. K. Siesio 1984 ; The density and distribution of ischemic'brain injury inthe rat f&lowing two to ten minutes of forebrain ischemia. Acta Neuropathol. Berl. ; 64: 319332. Stone, T. W., and N. R. Burton 1988 ; NMDA receptors and ligands in the vertebrate CNS. Prog. Neurobiol. 30: 333-368. Tavlor. M. D. G. Palmer. and A. S. Callahan 1984 ; Protective action by methylprednisolone, allopurinol and indbmethacin against stroke-induced damage to adenylate cyclase in gerbil cerebral cortex. Stroke 15: 329-335. Taylor, M. D., T. K. Mellert, J. L. Parmentier, and L. J. Eddy 1985 ; Pharmacological protection of reoxygenation damage to in vitro brain slice tissue. Brain Res. 347: 268-273. Turrens, J. F., A. Alexandre, and A. L. Lehninger 1985 ; Ubisemiquinone is the electron donor for superoxide formation by complex III of heart mithocondtia. Arch. Biochem. Biophys. 237: 408-414. Vassault, A. 1983 ; Lactate dehydrogenase. UV-method with pyruvate and NADH. In Methods of Enzymatic Analysis, Vol. 3: Enzymes I: Oxidoreductases, Transferuses, H. U. Bergmeyer, ed., pp. 118-126, Verlag Chemie, Weinheim, FRG. Wallenstein, S., C. L. Zucker, and J. L. Fleiss 1980 ; Some statistical methods useful in circulation research. Circ. Res. 47: l-t7. Westerberg, E., D. T. Monaghan, C. W. Cotman, and T. Wieloch 1987 ; Excitatory amino acid receptors and ischemic brain damage in the rat. Neurosci. Lett. 73: 119-124.
Intraocular hypertension, intraocular pressure, steroid, budesonide, fluticasone propionate, glaucoma, mometasone furoate, triamcinolone acetonide, 1133 intraocular pressure, intraocular hypertension, steroid, budesonide, fluticasone propionate, glaucoma, mometasone furoate, triamcinolone acetonide, 1133 intravascular hemolysis, acute kidney tubule necrosis, risedronic acid, drug induced disease, 744 intravenous anesthesia, adrenalin, breast feeding, bupivacaine, infraorbital nerve, lip reconstruction, nerve block, postoperative analgesia, postoperative pain, tramadol, respiration depression, 899 iodinated poppyseed oil, advanced cancer, chemoembolization, cisplatin, doxorubicin, liver cell carcinoma, lung embolism, mitomycin, adult respiratory distress syndrome, anemia, antineoplastic agent, chemotherapy induced emesis, cytostatic agent, drug fever, dyspnea, epistaxis, neutropenia, tachycardia, thrombocytopenia, 1240 iohexol, myelography, speech disorder, metrizamide, neurotoxicity, stuttering, 1286 irinotecan, breast cancer, colorectal cancer, docetaxel, estramustine, exemestane, lung non small cell cancer, pemetrexed, prostate cancer, aromatase inhibitor, arthralgia, bevacizumab, bleeding, cardiovascular disease, cetuximab, diarrhea, digestive system perforation, febrile neutropenia, fluorouracil, folinic acid, gastrointestinal symptom, hair loss, heart muscle ischemia, hypertension, infection, lung cancer, melanoma, muscle cramp, nausea, neutropenia, paresthesia, proteinuria, rash, skin tingling, tamoxifen, thromboembolism, thrombosis, uterus cancer, vagina bleeding, vomiting, 1197 - breast cancer, docetaxel, metastasis, alopecia, anemia, asthenia, blood toxicity, bone marrow toxicity, cardiotoxicity, diarrhea, drug fatality, fatigue, febrile neutropenia, liver toxicity, nausea, nephrotoxicity, neurotoxicity, neutropenia, thrombocytopenia, vomiting, 1246 - cancer chemotherapy, DNA topoisomerase inhibitor, rheumatoid arthritis, 1245 - cancer combination chemotherapy, colorectal cancer, cyclooxygenase 2 inhibitor, fluorouracil, metastasis, rofecoxib, alopecia, anemia, asthenia, blood toxicity, cardiotoxicity, celecoxib, cystitis, diarrhea, fever, folinic acid, gastrointestinal toxicity, granulocytopenia, hand foot syndrome, heart disease, heart infarction, hematologic disease, infection, leukopenia, liver toxicity, mucosa inflammation, nausea, nephrotoxicity, neutropenia, peripheral neuropathy, phlebitis, stomach pain, stroke, thrombocytopenia, thromboembolism, valdecoxib, vomiting, 1260 iron, anemia, iron deficiency, selenium, selenium deficiency, zinc, zinc deficiency, anaphylaxis, iron dextran, iron overload, 693 iron deficiency, anemia, iron, selenium, selenium deficiency, zinc, zinc deficiency, anaphylaxis, iron dextran, iron overload, 693 irradiation, endocrine ophthalmopathy, methylprednisolone, prednisolone, steroid, steroid therapy, acne, eye infection, gastrointestinal symptom, herpes zoster oticus, hot flush, hyperlipidemia, hypertension, insomnia, leukocytosis, moon face, nose infection, 1114 irritable colon, alosetron, biliary tract disease, bulking agent, cilansetron, diarrhea, disease exacerbation, ischemic colitis, ispagula, serotonin 3 antagonist, serotonin 4 agonist, tegaserod, 1096 - constipation, ischemic colitis, tegaserod, 1093 ischemic colitis, constipation, irritable colon, tegaserod, 1093 ischemic heart disease, diabetes mellitus, medical education, beta adrenergic receptor blocking agent, heart muscle ischemia, insulin resistance, sulfonylurea, 1165 - hypercholesterolemia, statine derivative, atorvastatin, cerivastatin, fluindostatin, hydroxymethylglutaryl coenzyme A reductase inhibitor, mevinolin, myopathy, pravastatin, rhabdomyolysis, simvastatin, 1187 - non insulin dependent diabetes mellitus, acarbose, anemia, Section 38 vol 41.2 and norvasc. The mean median ; ESR progressively fell from 18.5 24 ; range 5118 ; mm h pretreatment with RMAT, to 11.8 11 ; range 126 ; mm h at months P 0.009, paired Wilcoxon test ; . Correspondingly, the CRP fell from a mean median ; of 23.5 8.3 ; range 4.0109.0 ; mg L pretreatment, to 17.3 4.0 ; range 1.450.3 ; mg L at 18 months P 0.03 ; . A significant increase in serum albumin compared with the pretreatment value Table ; was observed at 12 and 18 months after commencement of RMAT, median value 40.0 g L P 0.05 ; and 40.5 g L P 0.04 ; , respectively.
Methylprednisolone may also be used to treat people who are not able to produce enough of their own corticosteroid naturally e, g and ortho and methylprednisolone.

A saturated aqueous solution has a ph of about 6 and is stable half-life over 20 years ; but stability decreases in acid or alkaline conditions, the paracetamol being slowly broken down into acetic acid and p-aminophenol. 11 d on mg d oral methylprednisolonne and 150 mg d AZA. After a remission period, which lasted throughout the summer, he presented to the Department of Medicine in September 2003 with complaints of weakness, weight loss, coughing and fever, the latter appearing every evening for a week. He did not cough up blood. He had 2-4 stools daily, without blood. He had no abdominal symptoms. His daily treatment on admission was 150 mg AZA, 16 mg methylprednisolone and doxycycline prescribed by his GP ; . The results of physical examinations cardiovascular and respiratory systems and abdomen ; were normal. Septic fever was present. Blood chemistry values were as follows: ESR 125 mm h, haematocrit 31%, hb 10.7 g L, white blood cell count 4.100 G L, platelets 325.000 G L, CRP 351 mg L, and uric acid 471 mol L. The haemoculture was negative and procalcitonin was 0.07 ng mL. The bacteriology pharynx, sputum and urine ; and virology CMV, EBV, Coxackie and adenovirus ; tests were negative. The chest X-ray 2003-09-09 ; revealed moderately increased interstitial shadowing with a ring-like consolidation in both lungs predominantly in the right ; , without cardiac or aortic abnormalities. The followup chest X-ray revealed progression Figure 1 ; . Despite the discontinuation of AZA, progressive interstitial inflammation was detected in both lungs. The patient was treated with 1 g d clarithromycin intravenously and 12 mg methylprednisolone orally. He was transferred to the intensive care unit because of the possibility of a severe opportunistic infection or an autoimmune disease. AZAassociated pneumonitis was also suspected. His respiratory failure was treated in the intensive care unit. Dyspnoea occurred on minor exertion, the patient had orthostatic hypotension, and he was weak. On physical examination, the liver was palpable 2 cm beyond the costal margin. Except for dyspnoea nothing abnormal was detected. Blood chemistry showed ESR 120 mm h, haematocrit 26%, hb 90 g L, white blood cell count 2.970-3.200 G L, platelets 426.000 G L, SGOT 246 U L, SGPT 91 U L, gamma GT 600 U L, LDH 1125 U L, and procalcitonin 0.07 ng L. Nasal oxygen and oxycodone.
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1. 2. 3. Guyton AC, Hall JE: Textbook of Medical Physiology, 9th Edition. Philadelphia: WB Saunders, 1996. Campbell IT, Baxter JN, Tweedie IE, Taylor GT, Keens SJ: IV fluids during surgery. Br J Anaesth 1990, 65: 726-729. Sweny P: Is postoperative oliguria avoidable? Br J Anaesth 1992, 67: 137-145. Can help. For example, a child who loves sport will take their medication if it helps them to perform better.




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